Home > Corpo > Carbossiterapia
Home > Corpo > Carbossiterapia
Carboxytherapy means the use of CO2 for therapeutic purposes. This method was used for the first time in France in 1932 for the treatment of peripheral arterial diseases.
Co2 is an odourless and colourless carbon dioxide gas. The subcutaneous administration of CO2 absolutely does not affect breathing and does not cause any risk of embolisms, indeed, the diffusibility of this gas is up to 30 times higher than that of oxygen.
Optical probe videocapillaroscopy and Doppler laser flowmetry are two methods of microangiological investigation that have shown respectively that the CO2 action subcutaneously is expressed through a mechanism of vasodilation and an increase in arteriolar sphygmicity with a consequent increase in flow velocity at the level of the microcirculation.
The mechanism of action consists, first of all, in a direct stimulation of the arteriolar smooth muscle cells (acidosis) and a sympathetic-mimetic action with a consequent increase in the flow rate and opening of the blocking devices at the level of the capillaries, and secondly in a reduction of the affinity of hemoglobin for O2 with increased tissue oxygenation and lipolysis.
Home > Corpo > Carbossiterapia
Carboxytherapy means the use of CO2 for therapeutic purposes. This method was used for the first time in France in 1932 for the treatment of peripheral arterial diseases.
Co2 is an odourless and colourless carbon dioxide gas. The subcutaneous administration of CO2 absolutely does not affect breathing and does not cause any risk of embolisms, indeed, the diffusibility of this gas is up to 30 times higher than that of oxygen.
Optical probe videocapillaroscopy and Doppler laser flowmetry are two methods of microangiological investigation that have shown respectively that the CO2 action subcutaneously is expressed through a mechanism of vasodilation and an increase in arteriolar sphygmicity with a consequent increase in flow velocity at the level of the microcirculation.
The mechanism of action consists, first of all, in a direct stimulation of the arteriolar smooth muscle cells (acidosis) and a sympathetic-mimetic action with a consequent increase in the flow rate and opening of the blocking devices at the level of the capillaries, and secondly in a reduction of the affinity of hemoglobin for O2 with increased tissue oxygenation and lipolysis.